Obesity is a growing unmet medical need in companion animals and a shared biological challenge across Animal Health and Human Pharma. Dogs and cats develop obesity under similar environmental pressures, yet they differ markedly in metabolic disease susceptibility and progression. For example, cats are particularly prone to obesity-associated diabetes mellitus, whereas dogs exhibit distinct patterns of insulin resistance, inflammatory responses, and obesity-related comorbidities. These differences are likely to reflect species-specific regulatory mechanisms operating across multiple tissues and organ systems, including adipose tissue, liver, pancreas, skeletal muscle, and immune compartments, as well as the endocrine and inflammatory networks that coordinate whole-body energy homeostasis.
While adipose tissue acts as a central endocrine and inflammatory organ, the liver and pancreas are critical regulators of glucose, lipid, and insulin homeostasis. Differences in adipose tissue signaling, hepatic metabolic flexibility, and pancreatic adaptation to chronic nutrient excess may contribute to the divergent susceptibility of dogs and cats to obesity-associated metabolic disease. Understanding the interplay between these tissues is therefore essential for identifying conserved disease drivers, species-specific modifiers, and robust therapeutic targets.
Rather than representing a limitation, these species-specific differences provide a powerful opportunity for translational target discovery. Dogs and cats can be viewed as naturally occurring variations of a shared metabolic disease system, where conserved biological pathways reveal fundamental drivers of obesity, while divergent molecular and cellular responses highlight mechanisms that influence disease susceptibility, target sensitivity, efficacy, and safety. Understanding why some regulatory networks remain conserved across species while others differ may help identify therapeutic targets that are both biologically central and resilient to biological variability.
The proposed postdoctoral research program within Boehringer Ingelheim's Animal Health Non-Infectious Diseases group should apply innovative approaches to suitable in vitro and ex vivo canine and feline models to identify conserved molecular drivers of obesity while systematically characterizing species-specific regulatory mechanisms.
By integrating molecular, functional, and network-level datasets across relevant metabolic tissues, the project aims to distinguish conserved driver networks from species-specific modifiers and thereby prioritize robust, pharmacologically actionable therapeutic targets and biomarkers.
The successful candidate will work within a highly collaborative and multidisciplinary environment where Animal Health laboratories are closely integrated with Human Pharma research teams, providing exposure to both cutting-edge biology and industry drug discovery processes.